Caustic ingestion can cause severe injury to the esophagus and the stomach. Alkali ingestions A STUDY OF CORROSIVE ESOPHAGITIS. Corrosive esophagitis usually occurs from accidental or suicidal ingestion of caustic substances (e.g. lye, household cleaners, bleaches, washing soda), and is. Esophagitis Secondary to Ingestion of Caustic Material. P. N. Symbas, M.D., S. E. Vlasis, B.S., and C. R. Hatcher, Jr., M.D.. ABSTRACT The records of

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In the absence of symptoms and in the presence of accidental ingestions especially those of less corrosive substancessignificant lesions are usually not esophagigis on upper endoscopy.

Published online May 6.

These traumas increase the scars, reducing even more the lumen of the organ. Biodegradable esophageal stent placement does not prevent high-grade stricture formation after circumferential mucosal resection in a porcine model. The upper gastrointestinal tract, on the other hand, may present as dysphagia or odynophagia for esophageal injury and hematemesis or epigastric pain for gastric involvement[ 78 ].

Experimental findings suggest that arteriolar and venular thrombosis with consequent ischemia may be more important than inflammation in the pathogenesis of acute corrosive injury[ 10 ].

Steroids in second degree caustic burns of the esophagus: Ewophagitis will present with rapid onset of odynophagia, chest pain, vomiting, and hematemesis. With findings of extensive damage and necrosis, aborting the procedure is not mandatory[ 78 ]. Ingestion of caustic or castic agents i. It is often associated with a lower incidence of stricture formation than gastric pull-up hence esophagiyis preferential use in the setting of a relatively spared and healthy stomach[ 16 ].


In the acute phase, esophagrams may show abnormal esophageal motility with diffuse spasms and poor primary peristalsis. Partial gastric resection is preferred by many[] for the long-term risk of malignant transformation, though the need for gastric resection as prophylaxis against future malignancy has been overstated in the literature[ 29 ].

In a study of children, Gundogdu et al. Prevention of stricture is still a debatable issue, yet esophageal stents may offer promising outcomes.

Caustic injury of the upper gastrointestinal tract: A comprehensive review

Laparoscopy can be very effective in reducing mortality rate for caustic ingestion in suicide attempt. Risk of esophageal cancer after caustic ingestion might be overestimated, yet endoscopic screening is still recommended. However, further studies are needed to establish the role esophagutis EUS in caustic injury[ 78 ].

Esophageal caistic studies report low amplitude and nonperistaltic contractions, with a significantly higher exposure to pH below 4, compared with control groups[ 16 causric. Sao Paulo Med J. Cerebral abscess complicating dilatation of a corrosive esophageal stricture. It usually caudtic the middle and lower third of the esophagus.

J Pediatr Surg ;6: One of the patients died a few hours after admission to the hospital due to organ necrosis. Edematous wall thickening with periesophageal soft tissue infiltration plus well-demarcated tissue interface. Appleton and Lange; Please review our privacy policy. Still a major surgical dilemma.


Management algorithm for caustic substance ingestion. Intraperitoneal injection of 5-fluorouracil has been effective in preventing strictures experimentally[ 86 ]. When the volume of the corrosive substance ingested is large, the entire stomach is scarred leading to a diffusely contracted stomach.

Management of esophageal caustic injury

This is due to the ready availability of these caustic agents as items of household use and loose regulatory control on its production. Sucralfate, bismuth compounds, substituted benzimidazoles, trimipramine and pirenzepine in the short- and long-term treatment of duodenal ulcer.

Loading Stack – 0 images remaining. Most patients who developed cancer were young when they ingested the caustic agent: Clinical spectrum and feeding practices.

Cancer is most commonly observed at the areas of anatomic narrowing, and may be related to increased exposure to the caustic substance. Therefore, after caustic injuries the placement of a nasogastric tube may be considered, but the decision should be made with caution and done on a case-by-case basis.

Gastric lavage and induced emesis are contraindicated for the risk of re-exposure to the corrosive agent and additional injury to the esophagus. This suggests that the statistically significant difference detected between sexes may have been related to the amounts of caustic agent ingested by men and women.

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